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Hypothalamic Inflammation and Glucose Regulation Explained

by | Last updated: Mar 7, 2024 | Hypothalamus | 0 comments

Does hypothalamus inflammation affect glucose regulation?

Let’s talk about it.

Your hypothalamus is key in glucose regulation. When your hypothalamus becomes inflamed, glucose regulation is altered. 

Inflammatory processes in the hypothalamus occur in two phases. First a transient, early inflammatory phase, and with sustained exposure to toxins like a high-fat diet. A secondary phase, in which prolonged inflammatory cascades lead to the activation of cellular stress mechanisms. 

Inflammation activates immune cells. 

Your brain has two types of immune cells. Like macrophages in the blood (imagine a little PacMan that eats invaders) – called astrocytes and microglia.

It has been noted in research that gluten as well as high-fat diets increase the number of these immune cells which destroy brain tissue. 

The hypothalamus plays a critical role in maintaining glucose homeostasis by regulating the pancreatic release of insulin and glucagon through its control over both branches of the autonomic nervous system. 

When the hypothalamus becomes inflamed, glucose is not regulated properly leading to insulin resistance, type 2 diabetes, obesity and metabolic syndromes. 

Therefore, supporting your hypothalamus, nutraceutically with Genesis Gold® can help optimize its function and maintain normal glucose metabolism. 

All in all, if you have any questions about hypothalamic inflammation and glucose regulation, please join my Hormone Reboot Training.

Hormone Reboot Training
References:

https://journals.physiology.org/doi/abs/10.1152/ajpcell.00533.2022#:~:text=The%20hypothalamus%20plays%20a%20critical,epinephrine%20from%20the%20adrenal%20glands.

About the Author - Deborah Maragopoulos FNP

Known as the Hormone Queen®️, I’ve made it my mission to help everyone – no matter their age – balance their hormones, and live the energy and joy their DNA and true destiny desires. See more about me my story here…

     

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